Boston, MA – If CO2 levels continue to rise as projected, the populations of 18 countries may lose more than 5% of their dietary protein by 2050 due to a decline in the nutritional value of rice, wheat, and other staple crops, according to new findings from Harvard T.H. Chan School of Public Health. Researchers estimate that roughly an additional 150 million people may be placed at risk of protein deficiency because of elevated levels of CO2 in the atmosphere. This is the first study to quantify this risk.
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Scientists at the University of California, Riverside investigating the composition of particulate matter(PM) and its sources at the Salton Sea have found that this shrinking lake in Southern California is exposing large areas of dry lakebed, called playa, that are acting as new dust sources with the potential to impact human health.
“Playas have a high potential to act as dust sources because playa surfaces often lack vegetation,” said Roya Bahreini, an associate professor of environmental sciences, who led the research project. “Dust emissions from playas increase airborne PM mass, which has been linked to cardiovascular disease, respiratory disease, and mortality.”
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Microbes that live in the gut don’t just digest food. They also have far-reaching effects on the immune system. Now, a new study shows that a particular gut microbe can prevent severe flu infections in mice, likely by breaking down naturally occurring compounds — called flavonoids — commonly found in foods such as black tea, red wine and blueberries.
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In order to reduce the cost of next-generation polymer electrolyte fuel cells for vehicles, researchers have been developing alternatives to the prohibitively expensive platinum and platinum-group metal (PGM) catalysts currently used in fuel cell electrodes. New work at Los Alamos and Oak Ridge national laboratories is resolving difficult fuel-cell performance questions, both in determining efficient new materials and understanding how they work at an atomic level. The research is described this week in the journal Science.
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The cold-loving fungus (Pseudogymnoascus destructans, or Pd) that causes white-nose syndrome, a disease that has killed millions of North American bats during hibernation, could also spread in summer months. Bats and humans visiting contaminated caves and mines can inadvertently contribute to the spread of the fungus, according to a recently published study by the U.S. Geological Survey.
USGS scientists tested samples collected from bats, the environment and equipment at eight bat hibernation sites in Kentucky, Indiana, Ohio, Tennessee and Virginia. They found that bats occupying such sites in summer can harbor the Pd fungus on their skin, and that Pd is more readily detectable in their guano, or feces.
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